index - Organisation de la cellule musculaire et thérapie de la myopathie centronucléaire autosomique dominante Accéder directement au contenu

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Cell signaling Autophagy cellular Duchenne muscular dystrophy DMD Myopathie Dynamin 2 Cytosquelette Cell proliferation Developmental myosin heavy chain Adult patients Actin Nucleus Coeur Domaine LEM Amphiphysin Cancer Autophagosome maturation Charcot-Marie-Tooth BAR proteins Allele specific RNA interference Allele‐specific silencing therapy Nuclear envelope Endocytosis Correlative microscopy Congenital myopathy Clathrin Disease heterogeneity Centronuclear myopathy Diaphragm Alpha-actinin-2 AAV RNA interference Gene therapy Neural crest cells Dystrophie musculaire de Duchenne Adhesion AAV8 Dystrophie musculaire d'Emery Dreifuss Dominant centronuclear myopathy DNM2 A-type lamins Dynamin Adeno-Associated virus Duchenne muscular dystrophy Caveolae Cavins Duchenne Muscular Dystrophy Muscular dystrophy Cell migration Autophagy Satellite cell ACTN2 Cross-presentation BAF CAV-3 gene Dynamin overexpression Disease modifiers Biophysics BMP signaling Ctdnep1 Adeno-associated virus Nesprin Myosin Animal models of human disease Cardiotoxin Cellular neuroscience Cytoskeleton Caveolins Atrial heart defects Skin AFM Clathrine Cellules de crête neurale Atrial cardiac defects Adeno-associated virus vector Caveolin DMyHC Allele-specific silencing therapy Outflow tract Lamin Cavéoles Core myopathy Cardiomyopathies Myopathy Dynamine Autosomal dominant centronuclear myopathy Muscle Allele-specific silencing Cross-bridge kinetics Migration Actin nucleus Autophagosome Mechanotransduction AD-CNM Becker muscular dystrophy BMD Developmental biology Biomarkers Skeletal muscle Dullard Antisense oligonucleotides CTL